The difference between the various Testosterone derivatives AKA Steroids

I have trying explain this for a very long time, and I know it must seem horribly repetitive to some, for that I am sorry

But I feel I need to keep repeating myself

There is zero evidence that the huge Androgenic:Anabolic Ratios we see in Rodent Models carry across to Human Use.

This is true in both Steroidal SARM and Non Steriodal SARMs

Its ” what else” they do other that Protein Accretion that determines what drugs we choose when and why

There are many factors from Personal Choice ( totally valid ) to Intra-individual Genetic Response ( this is truly truly profound )

From the drugs action on the Central Nervous System ( eg elevated aggression )

To the drugs effect as a Chemical Uncoupler ( eg Trenbolones action on the Progesterone Receptor in Brown Adipose Tissue agreed controversial but supportable )

To the degree of Anabolic response we can elicit balanced against consequential Toxicity at a particular dosage – eg Androl or SuperDrol – both great growth promoters but very harsh and just not suitable for sustainable longer multi years use

To the degree a particular drug causes secondary effects like increases in Gylocogen Synthase

and and and and and

All these things play a role in why we choose what drug when, rather than X drug in more “Anabolic” than Y drug .. we can do better than that in 2020.

Yes I agree you could even make an argument that says well if this drug makes me more aggressive in the Gym and I train harder I might grow faster, sure but that is the training that is causing the growth secondary to the drugs impact on Protein Accretion

That is the same as you saying Anavar ( one of only 4 FDA approved appetite stimulants ) makes me want to eat more I might grow faster, sure but that is the food that is causing the growth secondary to the drugs impact on Protein Accretion

Yes there is a difference between the various Testosterone derivatives, I mean ” hello” – one should not even need to explain this, yes that is why the different derivatives exist in the first place, they do different thing in clinical practice..

But

Its ” what else” they do other that Protein Accretion that determines what drugs we choose when and why

In clinical practice you dont choose Drug A over Drug B based on its more Anabolic or not..

No questions on this post please, I am not going to lie.. I am getting rather tired of having to be abused for educating guys for free..

I understand that if you just look at one day, its easy to say, brush it off, its one guy and that is true sure of course

But after 6 years of it.. its growing a little thin…

All I can think of is I need to do all this privately.. so those that are interested in understanding this can and the monkeys in the cheap seats cant even see what we are talking about.. let alone get in our way.

So no questions today please guys.. this post IMO stands by itself.. its not here to explain the detail, just the framework

Its ” what else” they do other than Protein Accretion that determines what drugs we choose when and why

Cheers

Victor Black

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